Forskningscentrum på Södersjukhuset/Karolinska Institutet  

SWEDISH DIABETES RESEARCH

Svenska | English  
   


   
  Förstasidan

Forskning

Publikationer

Presentation av forskningsgruppen


Kontakta oss
 
   

 

Svensk diabetesforskning i världsklass

Här presenterar vi information om den forskning vi bedriver på Södersjukhuset

Under de senaste åren har forskningen inom diabetes tagit stora steg i rätt riktning. Många människor är drabbade av denna sjukdom och här på Södersjukhuset i Stockholm bedrivs flera av de kanske mest framgångsrika projekt som finns i världen idag!

På denna webplats hittar du information om den diabetesforskning som pågår på Södersjukhuset. Informationen riktar sig dels till kollegor inom fältet men också till den som är intresserad av medicinsk forskning i allmänhet och diabetesforskning i synnerhet. 

Vill du vara med och stödja diabetesforskningen?

 

Södersjukhuset har en av Sveriges största diabetesmottagningar och bedriver också internationellt ledande diabetesforskning. Donationer till Södersjukhusets diabetesforskningsfond kan sättas in på plusgirokonto 1694817-6. Märk talongen ”diabetes”.

 

För framtiden

Läs mer om diabetes och diabetesforskning

 

 

 

 

 

 

 
 Events

 
 Senaste publikationer:

BLX-1002, a novel thiazolidinedione with no PPAR affinity, stimulates AMP-activated protein kinase activity, raises cytosolic Ca2+, and enhances glucose-stimulated insulin secretion in a PI3K-dependent manner
http://ajpcell.physiology.org/cgi/content/abstract/296/2/C346

Endocrine Aspects of Type II Diabetes
Molecular and Cellular Endocrinology, Special Issue: Endocrine Aspects of Type II Diabetes. Guest Editor: Professor Åke Sjöholm

Editorial foreword to MCE Special Issue on "Endocrine Aspects of Type II Diabetes Mellitus".


The potential beneficial role of glucagon-like peptide-1 in endothelial dysfunction and heart failure associated with insulin resistance
http://www.ncbi.nlm.nih.gov/pubmed/18792870?ordinalpos=4&itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pubmed_DefaultReportPanel.Pubmed_RVDocSum

Hypoglycemic pharmacological treatment of type 2 diabetes: Targeting the endothelium
http://www.ncbi.nlm.nih.gov/pubmed/19038307?ordinalpos=1&itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pubmed_DefaultReportPanel.Pubmed_RVDocSum

Pronounced elevation of resistin correlates with severity of disease in severe sepsis and septic shock
http://www.ncbi.nlm.nih.gov/pubmed/17452927?ordinalpos=16&itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pubmed_DefaultReportPanel.Pubmed_RVDocSum

C-reactive protein: a marker or a player?
http://www.ncbi.nlm.nih.gov/pubmed/17447899?ordinalpos=17&itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pubmed_DefaultReportPanel.Pubmed_RVDocSum

The potent vasodilator ethyl nitrite is formed upon reaction of nitrite and ethanol under gastric conditions
http://www.ncbi.nlm.nih.gov/pubmed/18482590?ordinalpos=7&itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pubmed_DefaultReportPanel.Pubmed_RVDocSum

Intravenous hydration with a 2.5% glucose solution in Type II diabetes.
http://www.ncbi.nlm.nih.gov/pubmed/16584385?ordinalpos=25&itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pubmed_DefaultReportPanel.Pubmed_RVDocSum

Current guidelines for the diagnosis of testosterone deficiency
http://www.ncbi.nlm.nih.gov/pubmed/19011285?ordinalpos=1&itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pubmed_DefaultReportPanel.Pubmed_RVDocSum

Regression of a large malignant gastrinoma on treatment with Sandostatin LAR: a case report
http://www.ncbi.nlm.nih.gov/pubmed/18376130?ordinalpos=2&itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pubmed_DefaultReportPanel.Pubmed_RVDocSum

Postprandial paradoxical IGFBP-1 response in obese patients with Type 2 diabetes
http://www.ncbi.nlm.nih.gov/pubmed/18208402?ordinalpos=3&itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pubmed_DefaultReportPanel.Pubmed_RVDocSum

High-fat diets: healthy or unhealthy?
http://www.ncbi.nlm.nih.gov/pubmed/17714072?ordinalpos=1&itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pubmed_DefaultReportPanel.Pubmed_RVDocSum

BLX-1002, a novel thiazolidinedione with no PPAR affinity, activates AMPK, raises cytosolic Ca2+, and enhances glucose- and PI3K-sensitive insulin release
http://www.ncbi.nlm.nih.gov/pubmed/19052259?ordinalpos=4&itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pubmed_DefaultReportPanel.Pubmed_RVDocSum

Growth hormone signaling in pancreatic beta-cells-Calcium handling regulated by growth hormone
http://www.ncbi.nlm.nih.gov/pubmed/18602447?ordinalpos=7&itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pubmed_DefaultReportPanel.Pubmed_RVDocSum

Proteomics in diabetes research
http://www.ncbi.nlm.nih.gov/pubmed/18657591?ordinalpos=2&itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pubmed_DefaultReportPanel.Pubmed_RVDocSum

Short-term glucocorticoid treatment increases insulin secretion in islets derived from lean mice through multiple pathways and mechanisms
http://www.ncbi.nlm.nih.gov/pubmed/18984029?ordinalpos=1&itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pubmed_DefaultReportPanel.Pubmed_RVDocSum

Diazoxide-induced beta-cell rest reduces endoplasmic reticulum stress in lipotoxic beta-cells
http://www.ncbi.nlm.nih.gov/pubmed/18644846?ordinalpos=3&itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pubmed_DefaultReportPanel.Pubmed_RVDocSum

Evaluation of the SELDI-TOF MS technique for protein profiling of pancreatic islets exposed to glucose and oleate
http://www.ncbi.nlm.nih.gov/pubmed/17661320?ordinalpos=5&itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pubmed_DefaultReportPanel.Pubmed_RVDocSum

Gender-specific regulation of pancreatic islet blood flow, insulin levels and glycaemia in spontaneously diabetic Goto-Kakizaki rats
http://www.ncbi.nlm.nih.gov/pubmed/18205625?ordinalpos=10&itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pubmed_DefaultReportPanel.Pubmed_RVDocSum

Ethanol acutely stimulates islet blood flow, amplifies insulin secretion, and induces hypoglycemia via nitric oxide and vagally mediated mechanisms
http://www.ncbi.nlm.nih.gov/pubmed/17916634?ordinalpos=14&itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pubmed_DefaultReportPanel.Pubmed_RVDocSum http://www.medicalnewstoday.com/articles/93141.php

Global expression profiling of glucose-regulated genes in pancreatic islets of spontaneously diabetic Goto-Kakizaki rats
http://www.ncbi.nlm.nih.gov/pubmed/17693612?ordinalpos=18&itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pubmed_DefaultReportPanel.Pubmed_RVDocSum

A busy cell--endoplasmic reticulum stress in the pancreatic beta-cell
http://www.ncbi.nlm.nih.gov/pubmed/17706338?ordinalpos=16&itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pubmed_DefaultReportPanel.Pubmed_RVDocSum

Vasoactive drugs enhance pancreatic islet blood flow, augment insulin secretion and improve glucose tolerance in female rats
http://www.ncbi.nlm.nih.gov/pubmed/17020539?ordinalpos=25&itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pubmed_DefaultReportPanel.Pubmed_RVDocSum

Attenuation of insulin secretion by insulin-like growth factor binding protein-1 in pancreatic beta-cells
http://www.ncbi.nlm.nih.gov/pubmed/17693389?ordinalpos=5&itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pubmed_DefaultReportPanel.Pubmed_RVDocSum

Gene expression regulated by pioglitazone and exenatide in normal and diabetic rat islets exposed to lipotoxicity
http://www.ncbi.nlm.nih.gov/pubmed/19065603?ordinalpos=3&itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pubmed_DefaultReportPanel.Pubmed_RVDocSum

Valid CSS! Valid HTML 4.01 Transitional ©Forskningscentrum på Södersjukhuset/Karolinska Institutet | Kontakta oss | Sitemap | Produktion: BTPS